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Home > Clinical cases Home > CASE 37: Acquired third nerve palsy
CASE 1: Congenital esotropia without nystagmus
CASE 2: Congenital esotropia with nystagmus, limited
CASE 3: Nystagmus blockage syndrome
CASE 4: Residual esotropia
CASE 5: Exotropia after surgery for esotropia
CASE 6: Exotropia after a slipped medial rectus muscle
CASE 7: Exotropia caused by a ‘lost’ medial rectus muscle
CASE 8: ‘V’ pattern exotropia with overaction
CASE 9: Dissociated vertical deviation (DVD)
CASE 10: ‘A’ esotropia after bimedial rectus recession
CASE 11: ‘A’ exotropia after bimedial rectus recession
CASE 12: Basic pattern intermittent exotropia
CASE 13: Divergence excess intermittent exotropia
CASE 14: Convergence insufficiency intermittent exotropia
CASE 15: Persistent diplopia after surgery for intermittent exotropia
CASE 16: Congenital Brown syndrome
CASE 17: Acquired Brown syndrome
CASE 18: Iatrogenic Brown syndrome
CASE 19: Duane syndrome with esotropia (class I)
CASE 20: Exotropic Duane syndrome with limited adduction
CASE 21: Duane syndrome with straight eyes and limited
CASE 22: Duane syndrome with simultaneous abduction
CASE 23: Class I superior oblique palsy
CASE 24: Class II acquired oblique palsy
CASE 25: Large-angle class III congenital superior
CASE 26: Large class IV acquired superior oblique palsy
CASE 27: Bilateral superior oblique palsy
CASE 28: Canine tooth syndrome: “class VII”
CASE 29: Congenital absence of the superior oblique tendon
CASE 30: Thyroid ophthalmopathy (Graves’ ophthalmopathy)
CASE 31: Thyroid ophthalmopathy (Graves’ ophthalmopathy)
CASE 32: Thyroid ophthalmopathy (Graves’ ophthalmopathy)
CASE 33: Unilateral sixth nerve palsy
CASE 34: Bilateral sixth nerve palsy
CASE 35: Bilateral sixth nerve palsy with persistent diplopia after realignment
CASE 36: Right sixth nerve palsy from intracranial aneurysm
CASE 37: Acquired third nerve palsy
CASE 38: Traumatic third nerve palsy with misdirection after successful horizontal alignment
CASE 39: Congenital third nerve palsy
CASE 40: Severe bilateral congenital third nerve palsy
CASE 41: Sensory exotropia
CASE 42: Residual sensory exotropia
CASE 43: Dissociated vertical deviation with true hypotropia (falling eye)
CASE 44: Double elevator palsy
CASE 45: Blowout fracture of the orbit
CASE 46: Acute blowout fracture
CASE 47: Congenital fibrosis syndrome
CASE 48: Möbius syndrome
CASE 49: Skew deviation with symptomatic diplopia
CASE 50: Acquired esotropia
CASE 51: Chronic progressive external ophthalmoplegia
CASE 52: Ocular myasthenia
CASE 53: Absence of the medial rectus muscle
CASE 54: Traumatic disinsertion of the inferior rectus muscle
CASE 55: Diplopia after cataract extraction from left inferior rectus restriction
CASE 56: Diplopia after repair of retinal detachment
CASE 57: Diplopia after repair of retinal detachment
CASE 58: ‘V’ pattern esotropia with overaction of the inferior oblique muscles
CASE 59: ‘A’ exotropia, bilateral overaction of the superior obliques, dissociated vertical deviation (DVD)
CASE 60: Parinuad’s paralysis of elevation
CASE 61: Null point nystagmus
CASE 62: Congenital nystagmus with decreased vision
CASE 63: Nystagmus after brain stem stroke
CASE 64: Superior oblique myokymia
CASE 65: Typical refractive esotropia
CASE 66: Refractive/accommodative esotropia (high AC/A)
CASE 67: Refractive esotropia with dissociated vertical deviation



CASE 37: Acquired third nerve palsy


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Clinical picture

Case 37

This patient has an acute right third nerve palsy. A, In the primary position, ptosis of the right upper lid is complete. B, With the right upper lid held up by the right patient’s finger, the right eye is exodeviated approximately 40 prism diopters and is slightly hypodeviated. C, Abduction of the right eye is full. D, The right eye cannot adduct even to the midline. E, On downgaze the right eye intorts slightly, suggesting unopposed superior oblique function.

History
This 28-year-old man sustained closed head trauma in a motor vehicle accident 13 months earlier. He is concerned because his right eye is closed. When he raises his right upper lid, he notes that his eye is deviated outward, and he sees double. He would like to have the right eye straightened and his right lid raised.

Examination
Complete ptosis of the right upper lid is present. With maximum attempt at elevation using the frontalis muscle, the right upper lid moves upward about 3 mm. Forty prism diopters of exotropia and 15 prism diopters of right hypotropia are present in the primary position. The right pupil is dilated to 6 mm and does not react to light or accommodative effort. Visual acuity is OD 20/30 and OS 20/20. This patient understands that if his eye is made straighter and his lid raised, he will continue to have double vision and that this double vision may be more bothersome because the images are closer. In spite of this, he would like to have surgery to improve alignment of his eyes.

Diagnosis
Traumatic right third nerve palsy.

Treatment/Surgery
Maximum recession of the right lateral rectus 10+ mm, right superior oblique tendon transfer without trochlea fracture or maximum recession of the right lateral rectus and 10+ mm resection of the right medial rectus with 1/2 to 3/4 muscle width upshift of both muscles.

Comment
The appropriate extraocular procedure, when successful, can align or nearly align the eye with third nerve palsy, but motility is always limited. When the lid is raised, postoperatively the involved eye during fixation in the primary position is usually slightly exotropic. Frontalis suspension of the upper lid can be performed at the same time as the extraocular muscle surgery or it can be done at a second procedure. Whenever it is done, the ptosis should be undercorrected to lessen the adverse effect of corneal exposure  which is the rule after frontalis lid suspension and limited upward protective movement (Bell phenomenon) of the eye. In a patient such as this, who lacks effective suppression, diplopia can be extremely bothersome. Actually, some of the most agitated and distraught patients I have treated have been of this category. This problem is especially severe when the patient with third nerve palsy is emotionally liable from brain injury. Patients with acquired third nerve palsy should be counseled thoroughly before surgery, telling them about the problems associated with postoperative diplopia. In several successfully aligned cases, it has been necessary to fit the patient with an occluding contact lens or to give glasses with an occluder lens to eliminate the diplopia. On the other hand, if suppression is present for any reason or if vision is poor in one eye alignment can be achieved and the patient is pleased. In some cases of complete acquired third nerve palsy it may be best to refrain from surgery and simply allow the ptosis to “treat” diplopia.

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