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Home > Section 2: Diagnostic and Treatment Decisions Home > 2.44 Congenital Absence of Superior Oblique Tendon
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2.01 Amblyopia: Diagnosis and Classification
2.02 Strabismic Amblyopia: Treatment
2.03 Anisometropic Amblyopia: Treatment
2.04 Essential Infantile Esotropia: Diagnostic Evaluation
2.05 Essential Infantile Esotropia: Treatment
2.06 Acquired Esotropia
2.07 Refractive Accommodative Esotropia: Etiology and Treatment
2.08 Convergence Excess Type Esotropia
2.09 Consecutive Esotropia
2.10 Acute Esotropia
2.11 Microtropia and Subnormal Binocular Vision
2.12 Exodeviations: Treatment
2.13 Consecutive Exotropia
2.14 Paralytic Cyclovertical Strabismus: Right Hypertropia
2.15 Paralytic Cyclovertical Strabismus: Left Hypertropia
2.16 Dissociated Deviations
2.17 Upshoot in Adduction: Right Eye
2.18 Upshoot in Adduction: Left Eye
2.19 Downshoot in Adduction: Right Eye
2.20 Downshoot in Adduction: Left Eye
2.21 Cyclotropia: Diagnosis
2.22 Cyclotropia: Treatment
2.23 A Pattern Strabismus: Treatment
2.24 V Pattern Strabismus: Treatment
2.25 Limitation of Elevation of One Eye
2.26 Limited Depression of One Eye
2.27 Limitation of Abduction
2.28 Limitation of Adduction
2.29 Limitation of Vertical Gaze of Both Eyes
2.30 Acquired Vertical Deviation With Diplopia
2.31 Generalized Limitation of Ocular Motility of Both Eyes
2.32 Painful Ophthalmoplegia
2.33 Compensation Strategies in Manifest Congenital Nystagmus
2.34 Compensation Strategies of Manifest-Latent Nystagmus
2.35 Treatment of Nystagmus
2.36 Complete Third Nerve Palsy
2.37 Superior Rectus Muscle Paralysis
2.38 Inferior Rectus Muscle Paralysis
2.39 Medial Rectus Muscle Paralysis
2.40 Inferior Oblique Muscle Paralysis
2.41 Fourth Nerve Paralysis: Classification
2.42 Superior Oblique Muscle Paralysis: Diagnosis
2.43 Superior Oblique Muscle Paralysis: Treatment
2.44 Congenital Absence of Superior Oblique Tendon
2.45 Sixth Nerve Paralysis: Diagnosis
2.46 Sixth Nerve Paralysis: Treatment
2.47 Divergence Insufficiency Versus Bilateral Abducens Paresis
2.48 Convergence Insufficiency
2.49 When to Use Prisms
2.50 Duane Syndrome Type I
2.51 Duane Syndrome Type II
2.52 Duane Syndrome Type III
2.53 Brown Syndrome
2.54 Orbital Floor Fracture
2.55 Endocrine Myopathy
2.56 Myasthenia Gravis
2.57 Child With Reading Problems
2.58 Muscle Reattachment Techniques
2.59 Advantages of General Versus Local Anesthesia
2.44 Congenital Absence of Superior Oblique Tendon

2.44

(1) This unexpected finding, which can also occur at the time of intended surgery on the superior oblique tendon (Harada-Ito procedure or tuck) can be suspected by observing marked to moderate facial asymmetry, associated horizontal strabismus, amblyopia, and underaction of the superior oblique muscle; however, it cannot be predicted reliably on the basis of preoperative findings and not infrequently occurs as a real surprise!  Nevertheless the surgeon must be prepared to encounter it and to consider alternative procedures if surgery on the superior oblique tendon is planned but the tendon cannot be located.24, p.487

(2) The primary procedure for superior oblique underaction is inferior oblique weakening, but this alone is usually insufficient treatment.

(3) Restricted passive infraduction is associated with apparent overaction of the contralateral superior oblique and is caused by contracture of the ipsilateral superior rectus muscle.71  If present, this sign calls for recession of the tight superior rectus.

(4) Inferior oblique overaction is one of the hallmarks of superior oblique paralysis or absence.  If this muscle has been weakened by previous surgery, the only alternative is to recess the contralateral inferior rectus muscle.

(5) Pure excyclotropia without vertical misalignment is infrequently encountered in connection with congenital absence of a superior oblique but has been observed by one of us (GKvN).  If the patient is symptomatic, horizontal transposition of the vertical rectus muscles should be considered68